60 Participants Needed

MitoQ for Cardiovascular Disease

(MOVHS Trial)

AT
ZJ
Overseen ByZach J Hutchison, MS
Age: 18+
Sex: Any
Trial Phase: Academic
Sponsor: Auburn University

Trial Summary

Will I have to stop taking my current medications?

The trial information does not specify whether you need to stop taking your current medications. However, it does mention that participants should be free from certain medical conditions and not have allergies to MitoQ.

What data supports the effectiveness of the drug MitoQ for cardiovascular disease?

Research on Coenzyme Q10 (CoQ10), a component similar to MitoQ, shows it can improve heart health by reducing blood pressure and improving heart function in people with heart failure. CoQ10 is known to help with energy production in cells and has antioxidant properties, which may help protect the heart from damage.12345

What makes the drug MitoQ unique for treating cardiovascular disease?

MitoQ is unique because it targets mitochondria directly, enhancing their function and reducing oxidative stress, which is different from other treatments that may not specifically focus on mitochondrial health. This approach can potentially improve energy production in heart cells and protect them from damage, offering a novel way to support cardiovascular health.13567

What is the purpose of this trial?

Cardiovascular diseases (CVDs) are the number one cause of death in America and most of the post-industrial world. Hypertension is a leading risk factor for CVDs including stroke, myocardial infarction, and heart failure. Black Americans suffer from the highest rates of hypertension of any racial/ethnic group in America, among the highest in the world. There are also well-documented racial disparities in vascular dysfunction (e.g., endothelial dysfunction, arterial stiffening). Thus, racial disparities in hypertension and vascular dysfunction exacerbate the burden of CVDs, with Black Americans being 30% more likely to die from CVD than any other race in the US. It is established that mitochondrial dysfunction contributes to vascular dysfunction. However, there is a knowledge gap regarding whether targeting mitochondrial dysfunction attenuates oxidative stress, vascular dysfunction, and CVD risk among Black adults at heightened CVD risk. Thus, the investigators will conduct an 8-week trial with the mitochondrial antioxidant MitoQ in middle-aged and older Black and non-Black adults. Our overarching hypothesis is that mitochondrial dysfunction contributes to heightened oxidative stress, vascular dysfunction, and higher BP in Black adults; and that MitoQ will attenuate these racial differences. Importantly, the investigators will also assess social determinants of health (e.g., income, neighborhood disadvantage, discrimination) and health behaviors (e.g., diet, physical activity) and uncover their role in oxidative stress, vascular function, and BP Regarding methodology, the investigators will perform blood draws, vascular testing, preceding and following an 8-week, 20mg daily consumption of MitoQ and placebo. The investigators will also measure urine biomarkers of kidney function and blood pressure in adults (45-75 years old).

Research Team

Loop | Austin T Robinson

Austin T Robinson, PhD

Principal Investigator

Kinesiology Building

Eligibility Criteria

This trial is for middle-aged and older adults (45-75 years old) who are generally healthy but at risk of cardiovascular disease, particularly Black Americans. Participants must not have medical conditions that prevent exercise, giving blood, or allergies to MitoQ ingredients. They should have a BMI under 40 Kg/m2 and blood pressure below 150/90 mmHg without a history of serious diseases like cancer, diabetes, or heart issues.

Inclusion Criteria

Have blood pressure no higher than 150/90 mmHg
I am between 45 and 75 years old.
I don't have health problems that stop me from exercising, giving blood, or taking MitoQ.
See 2 more

Exclusion Criteria

Known allergy to MitoQ
I have a history of kidney disease.
My BMI is over 40.
See 8 more

Timeline

Screening

Participants are screened for eligibility to participate in the trial

2-4 weeks

Baseline Assessment

Baseline assessments including blood draws, vascular testing, and questionnaires on sleep, mood, and social determinants of health

2 weeks
1-2 visits (in-person)

Treatment

Participants receive 8-week supplementation of MitoQ or placebo, with blood pressure and vascular function assessments

8 weeks
2 visits (in-person)

Follow-up

Participants are monitored for safety and effectiveness after treatment, including post-treatment blood draws and vascular testing

4 weeks
1 visit (in-person)

Treatment Details

Interventions

  • MitoQ
Trial Overview The study tests if the antioxidant MitoQ can reduce oxidative stress and improve vascular health over an 8-week period in individuals at risk for cardiovascular disease. It involves daily consumption of MitoQ compared to a placebo while also considering social factors and health behaviors.
Participant Groups
2Treatment groups
Experimental Treatment
Placebo Group
Group I: MitoQExperimental Treatment1 Intervention
8-week MitoQ supplementation (20 mg daily capsule).
Group II: PlaceboPlacebo Group1 Intervention
8-week placebo matched in appearance to MitoQ supplementation (20 mg daily capsule).

Find a Clinic Near You

Who Is Running the Clinical Trial?

Auburn University

Lead Sponsor

Trials
81
Recruited
14,600+

Findings from Research

CoQ10 supplementation has shown promise in improving various cardiovascular conditions, including congestive heart failure and ischemic heart disease, based on a review of 34 controlled trials and additional long-term studies.
Achieving higher blood levels of CoQ10 (over 3.5 micrograms/ml) through increased dosages is associated with greater clinical improvements, suggesting that CoQ10 may play a significant role in cardiovascular health and disease prevention.
Overview of the use of CoQ10 in cardiovascular disease.Langsjoen, PH., Langsjoen, AM.[2019]
CoQ10 supplementation may be beneficial as an adjunctive therapy for patients with congestive heart failure, particularly for those who cannot tolerate standard medical treatments, based on evidence from seven systematic reviews.
The reviews indicated improvements in key cardiovascular measures such as cardiac output and functional classification, suggesting that CoQ10 could play a role in managing cardiovascular diseases.
Coenzyme Q10 in the treatment of heart failure: A systematic review of systematic reviews.Jafari, M., Mousavi, SM., Asgharzadeh, A., et al.[2019]
Coenzyme Q10 (CoQ10) supplementation has been shown to reduce mortality and improve exercise capacity in patients with heart failure, highlighting its potential as an effective treatment for cardiovascular diseases.
CoQ10 can also lower blood pressure in hypertensive patients and protect heart function during chemotherapy, suggesting it may have multiple beneficial effects in cardiovascular health.
Efficacy of coenzyme Q10 in supportive therapy of the cardiovascular diseases and in the prevention of cardiotoxicity caused by chemotherapy.Miedziaszczyk, M., Cieślewicz, A., Jabłecka, A.[2022]

References

Overview of the use of CoQ10 in cardiovascular disease. [2019]
Coenzyme Q10 in the treatment of heart failure: A systematic review of systematic reviews. [2019]
Efficacy of coenzyme Q10 in supportive therapy of the cardiovascular diseases and in the prevention of cardiotoxicity caused by chemotherapy. [2022]
Usefulness of coenzyme Q10 in clinical cardiology: a long-term study. [2019]
Coenzyme Q10: Clinical Applications in Cardiovascular Diseases. [2020]
[Physiological aspects of ubiquinone supplementation in cardiovascular pathology]. [2006]
Co-enzyme Q10: a new drug for cardiovascular disease. [2019]
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