37 Participants Needed

CAR T Cell Therapy for Brain Cancer

DA
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Overseen ByBilal Omer, MD
No Placebo GroupAll trial participants will receive the active study treatment (no placebo)

Trial Summary

What is the purpose of this trial?

In this study, there are two treatment groups called Cohort 1 and Cohort 2. Cohort 1 is for patients with diffuse midline glioma, high grade glioma, diffuse intrinsic pontine glioma, medulloblastoma, or another rare brain cancer that expresses GD2. Cohort 2 is for patients with a type of cancer called progressive pontine diffuse midline glioma (DMG), high grade glioma or diffuse intrinsic pontine glioma that expresses GD2. Because there is no standard treatment at this time, patients are asked to volunteer in a gene transfer research study using special immune cells called T cells. T cells are a type of white blood cell that help the body fight infection. This research study combines two different ways of fighting cancer: antibodies and T cells. Both antibodies and T cells have been used to treat cancer patients. They have shown promise but have not been strong enough to cure most patients. Researchers have found from previous research that they can put a new antibody gene into T cells that will make them recognize cancer cells and kill them. GD2 is a protein found on several different cancers. Researchers testing brain cancer cells found that many of these cancers also have GD2 on their surface. In a study for neuroblastoma in children, a gene called a chimeric antigen receptor (CAR) was made from an antibody that recognizes GD2. This gene was put into the patients own T cells and given back to 11 patients. The cells did grow for a while but started to disappear from the blood after 2 weeks. The researchers think that if T cells are able to last longer they may have a better chance of killing tumor cells. In this study, a new gene will be added to the GD2 T cells that can cause the cells to live longer. T cells need substances called cytokines to survive. The gene C7R has been added that gives the cells a constant supply of cytokine and helps them to survive for a longer period of time. In other studies using T cells researchers found that giving chemotherapy before the T cell infusion can improve the amount of time the T cells stay in the body and therefore the effect the T cells can have. This is called lymphodepletion and it will allow the T cells to expand and stay longer in the body and potentially kill cancer cells more effectively. After treating 11 patients, the largest safe dose of GD2-CAR T cells given in the vein (IV) was determined. Going forward, we will combine IV infusions with infusions directly into the brain through the Ommaya reservoir or programmable VP shunt. The goal is to find the largest safe dose of GD2-C7R T cells that can be administered in this way. Patients will now be assigned to Cohort 1 and 2 based on their tumor type with different dose levels for each cohort. The GD2.C7R T cells are an investigational product not approved by the FDA. The purpose of this study is to combine infusions into the vein in the first treatment cycle with infusions directly into the cerebrospinal fluid (CSF) in the brain (intracerebroventricularly) through the ommaya reservoir or programmable VP shunt for the second infusion cycle and possibly additional infusions after that. The goal is to find the largest safe dose of GD2-C7R T cells that can be administered in this way, and additionally to evaluate how long they can be detected in the blood and CSF and what affect they have on brain cancer.

Do I need to stop my current medications to join the trial?

The trial requires you to stop any concurrent anti-cancer therapy at least three half-lives before treatment. You also cannot have received any other forms of immunotherapy within 42 days before the investigational agent or colony-stimulating factors within 14 days prior to lymphodepletion. The protocol does not specify about other medications, so please consult with the trial team for more details.

What data supports the idea that CAR T Cell Therapy for Brain Cancer is an effective treatment?

The available research shows that CAR T Cell Therapy targeting GD2 has shown promise in treating brain cancer. In one study, three out of four patients with a specific type of brain cancer showed improvement after receiving this treatment. Another study found that this therapy could effectively target and kill cancer cells in laboratory models of glioblastoma, a severe brain cancer. Additionally, when compared to other methods, delivering the treatment directly into the brain significantly increased survival time without side effects. These findings suggest that CAR T Cell Therapy could be a promising option for treating certain brain cancers.12345

What safety data is available for CAR T Cell Therapy targeting GD2 in brain cancer?

The safety data for GD2-targeted CAR T cell therapy in brain cancer is mixed. Studies indicate that GD2-specific CAR T cells have been generally well tolerated in clinical trials, with no dose-limiting toxicities reported in some cases. However, there are concerns about neurotoxicity, as seen in preclinical models where GD2 CAR T cells induced fatal neurotoxicity in a costimulatory domain-dependent manner. In another study, peritumoral neuroinflammation during antitumor activity led to lethal hydrocephalus in some animal models. Therefore, while the therapy shows promise, careful monitoring and management are necessary to mitigate potential neurotoxic effects.12678

Is the treatment (C7R)-GD2.CART cells promising for brain cancer?

Yes, the treatment (C7R)-GD2.CART cells is promising for brain cancer. It uses the body's immune system to target and destroy cancer cells, showing potential in treating aggressive brain tumors like glioblastoma and diffuse midline gliomas. Early trials have shown positive results in targeting specific proteins on tumor cells, and it could be transformative for certain childhood brain cancers.19101112

Research Team

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Bilal Omer, MD

Principal Investigator

Baylor College of Medicine

Eligibility Criteria

This trial is for children and young adults aged 1 to 21 with specific brain cancers that have a protein called GD2 on their surface. These include newly diagnosed or recurrent tumors like diffuse midline glioma, high-grade glioma, medulloblastoma, and others. Participants need measurable disease on MRI and a functional score indicating they can perform daily activities at least half the time.

Inclusion Criteria

My tumor is smaller than 5 cm.
Procurement
My tumor was between 5 and 5.5 cm but was surgically reduced in size.
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Timeline

Screening

Participants are screened for eligibility to participate in the trial

2-4 weeks

Lymphodepletion Chemotherapy

Patients receive cyclophosphamide and fludarabine (or clofarabine) to prepare for T cell infusion

3 days
1 visit (in-person)

T Cell Infusion and Monitoring

Patients receive GD2-C7R T cells via IV and/or ICV infusion and are monitored for side effects

4 weeks
Hospital stay for up to 5 days post-infusion, with monitoring in Houston for up to 4 weeks

Follow-up

Participants are monitored for safety and effectiveness after treatment

15 years
Visits at weeks 1, 2, 3, 4, 6, 8, then at months 3, 6, 9, 12, and biannually for 4 years, then annually

Treatment Details

Interventions

  • (C7R)-GD2.CART cells
Trial OverviewThe study tests genetically modified T cells (a type of immune cell) designed to last longer in the body by adding a gene called C7R. This helps them fight cancer more effectively. The trial involves two methods of infusing these cells: directly into the brain (ICV) and into the vein (IV), after chemotherapy to improve their survival.
Participant Groups
2Treatment groups
Experimental Treatment
Group I: C7R-GD2.CAR T cells (Cohort 2)Experimental Treatment1 Intervention
The dose level for autologous cell C7R-GD2.CAR T cell immunotherapy administered via intravenous (IV) infusion was determined in the initial phase of the protocol. The standard IV dosing is 10 million cells/m2 with lymphodepletion chemotherapy.
Group II: C7R-GD2.CAR T cells (Cohort 1)Experimental Treatment1 Intervention
The dose level for autologous cell C7R-GD2.CAR T cells administered via intravenous (IV) infusion was determined in the initial phase of the protocol. The standard IV dose is 10 million cells/m2 with lymphodepletion chemotherapy. In this subsequent phase of the study, the safe dosing levels for autologous cell C7R-GD2.CAR T cell immunotherapy administered intracerebroventricularly (ICV) via ommaya reservoir or programmable VP shunt in combination with subsequent IV doses will be determined.

Find a Clinic Near You

Who Is Running the Clinical Trial?

Baylor College of Medicine

Lead Sponsor

Trials
1,044
Recruited
6,031,000+

Faris Foundation

Collaborator

Trials
1
Recruited
40+

Center for Cell and Gene Therapy, Baylor College of Medicine

Collaborator

Trials
114
Recruited
2,900+

Violet Foundation for Pediatric Brain Cancer

Collaborator

Trials
1
Recruited
40+

The Faris Foundation

Collaborator

Trials
1
Recruited
40+

ChadTough Defeat DIPG Foundation

Collaborator

Trials
1
Recruited
40+

Findings from Research

GD2-targeted CAR T cells have shown promising efficacy in preclinical models of H3-K27M-mutant diffuse midline gliomas, effectively clearing tumors in five patient-derived xenograft models.
While the treatment was generally well tolerated, there were instances of neuroinflammation leading to hydrocephalus in some models, highlighting the need for careful monitoring and management in potential human trials.
Potent antitumor efficacy of anti-GD2 CAR T cells in H3-K27M+ diffuse midline gliomas.Mount, CW., Majzner, RG., Sundaresh, S., et al.[2022]
Infusion of GD2-specific fourth-generation safety-designed chimeric antigen receptor (4SCAR)-T cells in eight patients with GD2-positive glioblastoma (GBM) was found to be safe and well tolerated, with no severe adverse events reported.
Of the eight patients, four experienced a partial response lasting between 3 to 24 months, indicating that 4SCAR-T cells can exert anti-GBM activity, with a median overall survival of 10 months post-infusion.
Safety and antitumor activity of GD2-Specific 4SCAR-T cells in patients with glioblastoma.Liu, Z., Zhou, J., Yang, X., et al.[2023]
In a study involving patients treated with GD2-targeting CAR T cells, 75% experienced both radiographic and clinical improvements, indicating a strong efficacy of this treatment approach.
The results suggest that GD2-targeting CAR T cell therapy could be a promising option for patients, potentially leading to significant positive outcomes in their cancer treatment.
GD2-Targeting CAR T Cells Show Promise in H3K27M-Mutated Gliomas.[2022]

References

Potent antitumor efficacy of anti-GD2 CAR T cells in H3-K27M+ diffuse midline gliomas. [2022]
Safety and antitumor activity of GD2-Specific 4SCAR-T cells in patients with glioblastoma. [2023]
GD2-Targeting CAR T Cells Show Promise in H3K27M-Mutated Gliomas. [2022]
GD2 CAR T cells against human glioblastoma. [2021]
B7-H3 as a Novel CAR-T Therapeutic Target for Glioblastoma. [2020]
Preclinical assessment of the efficacy and specificity of GD2-B7H3 SynNotch CAR-T in metastatic neuroblastoma. [2021]
CAR T Cells Administered in Combination with Lymphodepletion and PD-1 Inhibition to Patients with Neuroblastoma. [2022]
Clinical Predictors of Neurotoxicity After Chimeric Antigen Receptor T-Cell Therapy. [2021]
Chimeric Antigen Receptor T-Cell Therapy: Updates in Glioblastoma Treatment. [2022]
10.United Statespubmed.ncbi.nlm.nih.gov
CAR T Cells. [2021]
11.United Statespubmed.ncbi.nlm.nih.gov
CAR T-cells to treat brain tumors. [2023]
12.United Statespubmed.ncbi.nlm.nih.gov
Future development of chimeric antigen receptor T cell therapies for patients suffering from malignant glioma. [2023]