Tapan M. Kadia | MD Anderson Cancer Center

Dr. Tapan Kadia, MD

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M D Anderson Cancer Center

Expert in Acute Myeloid Leukemia
Expert in Acute Myelogenous Leukemia
24 reported clinical trials
37 drugs studied

About Tapan Kadia, MD

Education:

  • Obtained MD from Baylor College of Medicine, Houston, Texas, in 2005.
  • Completed Residency in Internal Medicine at The Johns Hopkins Hospital, Baltimore, Maryland (2005-2008).
  • Undertook Fellowship in Hematology/Oncology at The University of Texas MD Anderson Cancer Center, Houston, Texas (2008-2011).

Experience:

  • Currently serves as a Professor of Medicine and Chief of the Section of Leukemia at The University of Texas MD Anderson Cancer Center.

Area of expertise

1Acute Myeloid Leukemia
Global Leader
Tapan Kadia, MD has run 16 trials for Acute Myeloid Leukemia. Some of their research focus areas include:
HLA
prior malignancy
myelodysplastic syndrome
2Acute Myelogenous Leukemia
Global Leader
Tapan Kadia, MD has run 13 trials for Acute Myelogenous Leukemia. Some of their research focus areas include:
HLA

Affiliated Hospitals

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M D Anderson Cancer Center
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The University Of Texas M.D. Anderson Cancer Center

Clinical Trials Tapan Kadia, MD is currently running

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Liposomal Daunorubicin-Cytarabine + Venetoclax

for Acute Myeloid Leukemia

This phase II trial studies how well liposome-encapsulated daunorubicin-cytarabine and venetoclax work in treating participants with acute myeloid leukemia that has come back (relapsed), does not respond to treatment (refractory), or has not been treated (untreated). Drugs used in chemotherapy, such as liposome-encapsulated daunorubicin-cytarabine and venetoclax, work in different ways to stop the growth of cancer cells, either by killing the cells, by stopping them from dividing, or by stopping them from spreading.
Recruiting1 award Phase 2
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Decitabine + Other Drugs

for Acute Myeloid Leukemia

This phase Ib trial is to find out the side effects and possible benefits of decitabine alone or given together with venetoclax, gilteritinib, enasidenib, or ivosidenib in treating patients with acute myeloid leukemia that is under control (remission). Chemotherapy drugs, such as decitabine, work in different ways to stop the growth of cancer cells, either by killing the cells, by stopping them from dividing, or by stopping them from spreading. Venetoclax may stop the growth of cancer cells by blocking a protein called Bcl-2 needed for cell growth. Gilteritinib, enasidenib, and ivosidenib may stop the growth of cancer cells by blocking some of the enzymes needed for cell growth. Giving decitabine alone or together with venetoclax, gilteritinib, enasidenib, or ivosidenib may help to control the disease.
Recruiting1 award Phase 114 criteria

More about Tapan Kadia, MD

Clinical Trial Related6 years of experience running clinical trials · Led 24 trials as a Principal Investigator · 9 Active Clinical Trials
Treatments Tapan Kadia, MD has experience with
  • Venetoclax
  • Decitabine
  • Cladribine
  • Cytarabine
  • APG-2575
  • Azacitidine

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