Edward J. Kim, M.D., Ph.D. for UC Davis ...

Dr. Edward J. Kim

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University of California Davis Comprehensive Cancer Center

Studies Pancreatic Cancer
Studies Liver Cancer
33 reported clinical trials
70 drugs studied

About Edward J. Kim

Education:

  • Earned an A.B. in Chemistry from Harvard University, Cambridge, MA (1997).
  • Received M.D. and Ph.D. from Stony Brook University, Stony Brook, NY (2005, 2003 respectively).

Experience:

  • Completed Internal Medicine Residency and Hematology/Oncology Fellowship at the University of Michigan, Ann Arbor, MI (2005-2011).
  • Serves as Medical Director at UC Davis Comprehensive Cancer Center’s Office of Clinical Research.
  • Holds the position of Associate Professor of Medicine, specializing in gastrointestinal malignancies.
  • Actively involved in clinical and laboratory research, focusing on novel treatments and patient-derived models for pancreatic cancer.

Area of expertise

1Pancreatic Cancer
Edward J. Kim has run 9 trials for Pancreatic Cancer. Some of their research focus areas include:
Stage IV
Stage III
PD-L1 positive
2Liver Cancer
Edward J. Kim has run 6 trials for Liver Cancer. Some of their research focus areas include:
Stage IV
Stage III
PD-L1 positive

Affiliated Hospitals

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University Of California Davis Comprehensive Cancer Center
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Fremont - Rideout Cancer Center

Clinical Trials Edward J. Kim is currently running

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Chemotherapy + Hormone Therapy

for Breast Cancer

This trial is testing if adding additional cancer-fighting drugs to treatments that stop certain body functions is better than just using the treatments that stop those functions alone. It targets younger women with early-stage breast cancer who have a higher risk of dying from the disease. The treatment works by stopping certain body functions and using drugs to kill cancer cells.
Recruiting2 awards Phase 319 criteria
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Triapine + Targeted Radiation

for Neuroendocrine Cancer

This phase II trial compares the effect of adding triapine to lutetium Lu 177 dotatate versus lutetium Lu 177 dotatate alone (standard therapy) in shrinking tumors or slowing tumor growth in patients with neuroendocrine tumors that have spread from where they first started (primary site) to other places in the body (metastatic). Triapine may stop the growth of tumor cells by blocking some of the enzymes needed for deoxyribonucleic acid synthesis and cell growth. Lutetium Lu 177 dotatate is a radioactive drug. It binds to a protein called somatostatin receptor, which is found on some neuroendocrine tumor cells. Lutetium Lu 177 dotatate builds up in these cells and gives off radiation that may kill them. It is a type of radioconjugate and a type of somatostatin analog. Giving triapine in combination with lutetium Lu 177 dotatate may be more effective at shrinking tumors or slowing tumor growth in patients with metastatic neuroendocrine tumors than the standard therapy of lutetium Lu 177 dotatate alone.
Recruiting1 award Phase 2

More about Edward J. Kim

Clinical Trial Related2 years of experience running clinical trials · Led 33 trials as a Principal Investigator · 15 Active Clinical Trials
Treatments Edward J. Kim has experience with
  • Nivolumab
  • Cisplatin
  • Gemcitabine
  • Nab-paclitaxel
  • Radiation Therapy
  • Hypofractionated Radiation Therapy

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