Triac for Allan-Herndon-Dudley Syndrome

Not currently recruiting at 7 trial locations
WV
Fv
Overseen ByF.S. van Geest, MD
Age: < 18
Sex: Male
Trial Phase: Phase 2
Sponsor: Rare Thyroid Therapeutics International AB
No Placebo GroupAll trial participants will receive the active study treatment (no placebo)
Prior Safety DataThis treatment has passed at least one previous human trial

What You Need to Know Before You Apply

What is the purpose of this trial?

This trial explores a treatment called Triac (a thyroid hormone analog) for young boys with Allan-Herndon-Dudley syndrome (AHDS), a rare condition. An issue with the MCT8 gene causes AHDS, leading to brain development problems and an overactive thyroid in other body parts. The trial will assess whether Triac can improve these symptoms after nearly two years of treatment. Boys under 30 months old with a known MCT8 gene mutation, who haven't received certain thyroid medications for extended periods, are suitable candidates for this trial. As a Phase 2 trial, this research measures the treatment's effectiveness in an initial, smaller group, offering a chance to contribute to important early findings.

Will I have to stop taking my current medications?

If your child has been treated with LT4 or PTU for less than three months, they can join the study six weeks after stopping these medications. If they have been on these medications for more than three months, they cannot participate.

Is there any evidence suggesting that Triac is likely to be safe for humans?

Research has shown that Triac, a treatment under study for Allan-Herndon-Dudley syndrome (AHDS), appears safe. In a study with children and adults with MCT8 deficiency, the same condition targeted by this trial, Triac was generally well-tolerated, with no major side effects directly linked to the treatment.

In these previous cases, Triac helped lower high levels of thyroid hormone, a key issue in AHDS. While it didn't resolve all problems, its safety profile supports further exploration of its benefits.

Triac is currently in a phase 2 trial, indicating it has already passed initial safety tests. This phase focuses more on the treatment's effectiveness, but safety remains closely monitored. So far, the safety data has been encouraging.12345

Why do researchers think this study treatment might be promising?

Unlike the standard of care for Allan-Herndon-Dudley Syndrome, which primarily focuses on managing symptoms, Triac directly targets the underlying problem. This condition is tied to a deficiency in the MCT8 protein, which affects thyroid hormone transport. Triac is a thyroid hormone analog that can bypass this transport issue, potentially correcting the hormone imbalance. Researchers are excited because this could directly address the root cause of the syndrome rather than just alleviating symptoms, offering a more effective treatment option.

What evidence suggests that Triac might be an effective treatment for Allan-Herndon-Dudley syndrome?

Research has shown that Triac, the treatment under study for MCT8 deficiency, also known as Allan-Herndon-Dudley syndrome (AHDS), may help manage symptoms of peripheral thyrotoxicosis, where the body's tissues overreact to thyroid hormones. Studies suggest that Triac can slow the loss of developmental skills in young patients, helping them maintain progress. However, while Triac has been effective for some symptoms, it does not appear to improve thyroid hormone function in the brain. Overall, Triac may offer important benefits for managing certain aspects of MCT8 deficiency.14678

Who Is on the Research Team?

Andrew Bauer, MD | Neurosurgeon in ...

Andrew Bauer, MD

Principal Investigator

Children's Hospital of Philadelphia

WV

W.E. Visser, MD, PhD

Principal Investigator

Erasmus Medical Center

KS

Kristina Sjöblom Nygren, MD

Principal Investigator

Rare Thyroid Therapeutics International AB

SL

Stephen LaFranchi_Nicol

Principal Investigator

Oregon Health& Science University (OHSU) Doernbecher Childrens Hospital

JL

Jan Lebl

Principal Investigator

Charles University and Motol University Hospital

HK

Heiko Krude

Principal Investigator

Charité - Universitätsmedizin Berlin Institut fur experimental paediatrische endokrinologie

Are You a Good Fit for This Trial?

This trial is for young boys aged 0 to 30 months with MCT8 deficiency, also known as Allan-Herndon-Dudley Syndrome. They must have a mutation in the MCT8 gene and their parents or guardians should agree to follow study procedures. Boys who've had certain thyroid treatments or other investigational drugs recently are not eligible.

Inclusion Criteria

I am a male with a confirmed MCT8 gene mutation.
I am between 0 and 30 months old.
Signed and dated informed consent form from the parents or legal guardian
See 1 more

Exclusion Criteria

I have been treated with thyroid medication for more than 3 months or stopped it at least 6 weeks ago with stable tests.
I am not allergic to the trial medication and do not have issues digesting lactose.
Treatment with another investigational drug or participation in other interventional trial within three months prior to baseline visit 1.
See 2 more

Timeline for a Trial Participant

Screening

Participants are screened for eligibility to participate in the trial

2-4 weeks

Treatment

Participants receive tiratricol treatment to evaluate its effect on neurodevelopment impairment and peripheral thyrotoxicosis

96 weeks
Regular visits for monitoring and assessment

Follow-up

Participants are monitored for safety and effectiveness after treatment

4 weeks

Long-term treatment

Participants may continue treatment for an additional 2 years to evaluate long-term effects

2 years

What Are the Treatments Tested in This Trial?

Interventions

  • Triac
Trial Overview The Triac Trial II studies the effects of tiratricol (Triac) on brain hypothyroidism and peripheral hyperthyroidism in patients with MCT8 deficiency. The treatment lasts for 96 weeks, assessing its impact on neurodevelopmental impairment, with an option to continue for two more years.
How Is the Trial Designed?
1Treatment groups
Experimental Treatment
Group I: MCT8 deficiency patientsExperimental Treatment1 Intervention

Find a Clinic Near You

Who Is Running the Clinical Trial?

Rare Thyroid Therapeutics International AB

Lead Sponsor

Trials
4
Recruited
60+

Erasmus Medical Center

Collaborator

Trials
742
Recruited
2,156,000+

Citations

Effectiveness and safety of Triac in children and adults with ...Effectiveness and safety of Triac in children and adults with MCT8 deficiency: an international, multicentre, single group, open-label, phase 2 trial
Impact of Early Intervention with Triiodothyroacetic Acid on ...This case presents evidence that Triac may resolve peripheral thyrotoxicosis successfully and may slow neurodevelopmental regression, while some developmental ...
Long-Term Efficacy of T3 Analogue Triac in Children and ...Long-term efficacy of T3 analogue triac in children and adults with MCT8 deficiency: a real-life retrospective cohort study.
TRIAC disrupts cerebral thyroid hormone action via ...TRIAC administration does not upregulate thyroid hormone action in the cerebrum. TRIAC is not efficiently trafficked into the cerebrum but suppresses the HPT ...
Thyroid Hormone Analog Therapy in MCT8 Deficiency ...Patients were assessed for study outcomes at baseline and 12 months after starting Triac administration. In the interval, patients were evaluated and screened ...
Effectiveness and safety of the tri-iodothyronine analogue ...The tri-iodothyronine (T3) analogue Triac (3,3',5-tri-iodothyroacetic acid) was reported to completely prevent the neurological signs in mouse ...
3,3 ,5-Triiodothyroacetic acid = 90 51-24-1Thyroid hormone research: A study explored the impact of 3,3′,5-Triiodothyroacetic acid on peripheral and neurodevelopmental findings in patients with MCT8 ...
Allan-Herndon-Dudley syndromeThe T3 analogue tiratricol (3,5,3'-triiodothyroacetic acid; TRIAC) is able to reduce T3 concentrations and to improve different features (e.g. low body ...
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